Seite - 60 - in Advances in Neuroimmunology
Bild der Seite - 60 -
Text der Seite - 60 -
BrainSci. 2016,6, 41
5. Stefanovic, A.N.; Stöckl, M.T.; Claessens, M.M.; Subramaniam, V.α-Synuclein oligomers distinctively
permeabilizecomplexmodelmembranes.FEBSJ.2014,281, 2838â2850. [CrossRef] [PubMed]
6. Danzer,K.M.;Haasen,D.;Karow,A.R.;Moussaud,S.;Habeck,M.;Giese,A.;Kretzschmar,H.;Hengerer,B.;
Kostka,M.Differentspeciesofalpha-synucleinoligomers inducecalciuminïŹuxandseeding. J.Neurosci.
2007,27, 9220â9232. [CrossRef] [PubMed]
7. Luth,E.S.; Stavrovskaya, I.G.;Bartels,T.;Kristal,B.S.; Selkoe,D.J. Soluble,preïŹbrillarα-synucleinoligomers
promotecomplexI-dependent,Ca2+-inducedmitochondrialdysfunction. J.Biol. Chem.2014,289, 21490â21507.
[CrossRef] [PubMed]
8. Volpicelli-Daley, L.A.; Gamble, K.L.; Schultheiss, C.E.; Riddle, D.M.; West, A.B.; Lee, V.M. Formation
of α-synuclein Lewy neurite-like aggregates in axons impedes the transport of distinct endosomes.
Mol. Biol. Cell2014,25, 4010â4023. [CrossRef] [PubMed]
9. Lashuel, H.A.; Petre, B.M.; Wall, J.; Simon,M.; Nowak, R.J.; Walz, T.; Lansbury, P.T. Alpha-synuclein,
especially theParkinsonâs disease-associatedmutants, formspore-like annular and tubularprotoïŹbrils.
J.Mol. Biol.2002,322, 1089â1102. [CrossRef]
10. Hurtig,H.I.; Trojanowski, J.Q.;Galvin, J.; Ewbank,D.; Schmidt,M.L.;Lee,V.M.;Clark,C.M.;Glosser,G.;
Stern, M.B.; Gollomp, S.M.; et al. Alpha-synuclein cortical Lewy bodies correlate with dementia in
Parkinsonâsdisease.Neurology2000,54, 1916â1921. [CrossRef] [PubMed]
11. Tanaka, M.; Kim, Y.M.; Lee, G.; Junn, E.; Iwatsubo, T.; Mouradian, M.M. Aggresomes formed by
alpha-synucleinandsynphilin-1arecytoprotective. J.Biol. Chem. 2004,279, 4625â4631. [CrossRef] [PubMed]
12. Reeve,A.;Simcox,E.;Turnbull,D.AgeingandParkinsonâsdisease:Whyisadvancingagethebiggest risk
factor?AgeingRes. Rev. 2014,14, 19â30. [CrossRef] [PubMed]
13. Hirsch,E.;Graybiel,A.M.;Agid,Y.A.Melanizeddopaminergicneuronsaredifferentially susceptible to
degeneration inParkinsonâsdisease.Nature1988,334, 345â348. [CrossRef] [PubMed]
14. Dias,V.; Junn,E.;Mouradian,M.M.The roleofoxidative stress inParkinsonâsdisease. J. ParkinsonsDis.
2013,3, 461â491. [PubMed]
15. Bharath,S.;Hsu,M.;Kaur,D.;Rajagopalan,S.;Andersen, J.K.Glutathione, ironandParkinsonâsdisease.
Biochem.Pharmacol. 2002,64, 1037â1048. [CrossRef]
16. Venkateshappa, C.; Harish, G.; Mythri, R.B.; Mahadevan, A.; Bharath, M.M.; Shankar, S.K. Increased
oxidativedamageanddecreasedantioxidantfunctioninaginghumansubstantianigracomparedtostriatum:
Implications forParkinsonâsdisease.Neurochem.Res. 2012,37, 358â369. [CrossRef] [PubMed]
17. Paolicelli, R.C.; Gross, C.T. Microglia in development: Linking brain wiring to brain environment.
NeuronGliaBiol.2011,7, 77â83. [CrossRef] [PubMed]
18. Schafer,D.P.;Lehrman,E.K.;Kautzman,A.G.;Koyama,R.;Mardinly,A.R.;Yamasaki,R.;Ransohoff,R.M.;
Greenberg,M.E.; Barres, B.A.; Stevens, B.Microglia sculpt postnatal neural circuits in an activity and
complement-dependentmanner.Neuron2012,74, 691â705. [CrossRef] [PubMed]
19. Jurgens, H.A.; Johnson, R.W. Dysregulated neuronal-microglial cross-talk during aging, stress and
inïŹammation.Exp.Neurol. 2012,233, 40â48. [CrossRef] [PubMed]
20. Lee,D.C.;Ruiz,C.R.;Lebson,L.;Selenica,M.-L.B.L.;Rizer, J.;Hunt, J.B.;Rojiani,R.;Reid,P.;Kammath,S.;
Nash,K.; etal.Agingenhancesclassicalactivationbutmitigatesalternativeactivation in thecentralnervous
system.Neurobiol. Aging2013,34, 1610â1620. [CrossRef] [PubMed]
21. Norden,D.M.;Muccigrosso,M.M.;Godbout, J.P.Microglialprimingandenhancedreactivity tosecondary
insult inaging,andtraumaticCNSinjury,andneurodegenerativedisease.Neuropharmacology2015,96, 29â41.
[CrossRef] [PubMed]
22. Hickman, S.E.; Kingery, N.D.; Ohsumi, T.K.; Borowsky, M.L.; Wang, L.C.; Means, T.K.; El Khoury, J.
The microglial sensomerevealedbydirectRNAsequencing.Nat.Neurosci. 2013,16, 1896â1905. [CrossRef]
[PubMed]
23. Cho,S.-H.H.;Chen,J.A.;Sayed,F.;Ward,M.E.;Gao,F.;Nguyen,T.A.;Krabbe,G.;Sohn,P.D.;Lo,I.;Minami,S.;
et al. SIRT1deïŹciency inmicroglia contributes to cognitivedecline inagingandneurodegenerationvia
epigenetic regulationof IL-1ÎČ. J.Neurosci. 2015,35, 807â818. [CrossRef] [PubMed]
24. McGeer, P.L.; Itagaki, S.; Akiyama,H.;McGeer, E.G.Rate of cell death inparkinsonism indicates active
neuropathologicalprocess.Ann.Neurol. 1988,24, 574â576. [CrossRef] [PubMed]
25. Knott, C.; Stern, G.; Wilkin, G.P. InïŹammatory regulators in Parkinsonâs disease: iNOS, lipocortin-1,
and cyclooxygenases-1and-2.Mol.Cell.Neurosci. 2000,16, 724â739. [CrossRef] [PubMed]
60
zurĂŒck zum
Buch Advances in Neuroimmunology"
Advances in Neuroimmunology
- Titel
- Advances in Neuroimmunology
- Autor
- Donna Gruol
- Herausgeber
- MDPI
- Ort
- Basel
- Datum
- 2017
- Sprache
- englisch
- Lizenz
- CC BY-NC-ND 4.0
- ISBN
- 978-3-03842-571-7
- Abmessungen
- 17.0 x 24.0 cm
- Seiten
- 164
- Schlagwörter
- neuroimmune, cytokine, chemokine, glia cel, neuron, neurodevelopment, neuroimmune disorder, neurologic disease, psychiatric disease, neuronal injury
- Kategorie
- Medizin